![]() Bladder outlet obstruction (e.g., BPH ).Neuropathy and polyuria in diabetes mellitus.Neurogenic bladd er in multiple sclerosis.Impaired (weak) detrusor contractility due to:.Overflow incontinence ( overflow bladder ) Long-term management: usually surgical (e.g., fistula repair), in consultation with urology and/or urogynecology.Short-term management: pads and external catheters.Urinary leakage occurs at all times, with no associated preceding symptoms or specific trigger activity.Complete loss of sphincter function (due to previous surgery, nerve damage, metastasis) or abnormal anatomy ( fistula between urinary tract and skin).Treat the most bothersome symptom first, e.g., anticholinergics for urge incontinence.May have any of the clinical features above.Combination of mechanisms of stress incontinence and urge incontinence.See “ Treatment of urge incontinence” for additional information.Second line: interventional procedures (e.g., sacral nerve stimulation, injection of botulinum toxin into the bladder wall).Strong, sudden sense of urgency, followed by involuntary leakage.Inflammatory conditions (e.g., UTI ) or neurogenic disorders → sphincter dysfunction, detrusor overactivity, or overactive bladder → autonomous contractions of the detrusor muscle and premature initiation of a normal micturition reflex.See “ Treatment of stress incontinence” for additional information.Surgical procedures (e.g., urethral slings or suspensions, artificial urinary sphincter).Injection of periurethral bulking agents.Minimally-invasive solutions, e.g., vaginal pessaries or urethral inserts.In refractory or severe incontinence, refer to urology for:.Trial of conservative management of UI for 6–8 weeks.Positive bladder stress test : urinary leakage during activities that increase intraabdominal pressure (e.g., coughing, Valsalva maneuver).Increase in intraabdominal pressure (e.g., from laughing, sneezing, coughing, exercising) → ↑ pressure within the bladder → bladder pressure > urethral sphincter resistance to urinary flow.Intrinsic sphincter deficiency, caused by:.Childbirth (i.e., damage of the pelvic floor muscle levator ani and/or the S2 – S4 nerve roots ).Poor pelvic support caused by pelvic postmenopausal estrogen loss.Urethral hypermobility in women ( bladder outlet incompetence ) secondary to:. ![]() To remember the reversible causes of acute urinary incontinence, think DIAPPERS: Delirium/confusion, Infection, Atrophic urethritis/ vaginitis, Pharmaceutical, Psychiatric causes (especially depression), Excessive urinary output ( hyperglycemia, hypercalcemia, CHF), Restricted mobility, Stool impaction. Excessive urinary output (in conditions like hyperglycemia, hypercalcemia, CHF).Psychiatric causes (especially depression, delirium/confused state).Transient causes of urinary incontinence.If left untreated, UI can have a severely detrimental effect on patients' psychosocial well-being, mobility, and independence, and can increase the risk of infection.įor the management of stress incontinence and urge incontinence, see also the respective articles. Initial management involves conservative measures (e.g., management of comorbidities, pelvic floor exercises, bladder training) and provision of continence products further treatment is based on the underlying mechanism and may involve pharmacotherapy or surgery. Advanced diagnostic studies may be required for patients with red flags in urinary incontinence or incontinence refractory to treatment. ![]() The diagnosis can often be made based on a detailed medical history, a voiding diary, physical examination, and basic testing including urinalysis and measurement of postvoid residual volume ( PVR). UI is more common in older individuals, and approximately twice as common in women than in men. Stress incontinence, urge incontinence, and mixed incontinence are the most common types. ![]() Urinary incontinence (UI) is a common condition characterized by involuntary leakage of urine.
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